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-Equilibrium Binding

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-Lambda Phage

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-Synthetic Genetic Switches

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-Stability Analysis

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-Fine-tuned versus Robust Chemotaxis Models

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-Wrapping up Chemotaxis

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-Genetic Oscillators

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-Genetic Oscillators (cont.)

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- In cultures of dissociated rat hippocampal neurons, persistent potentiation and depression of glutamatergic synapses were induced by correlated spiking of presynaptic and postsynaptic neurons.

- Long-term protestation of synaptic transmission in the hippocampus is the primary experimental model for investigating the synaptic basis of learning and memory in vertebrates.

- Activity-driven modifications in synaptic connections between neurons in the neocortex may occur during development and learning.

- Early visual experience is essential in the refinement of developing neural connections. In vivo whole-cell recording from the tectum of Xenopus tadpoles showed that repetitive dimming-light stimulation applied to the contralateral eye resulted in persistent enhancement of glutamatergic inputs, but not GABAergic or glycinergic inputs, on tectal neurons.                

- Calcium ions play crucial signaling roles in many forms of activity-dependent synaptic plasticity.

- Long-term potentiation (LTP) is an enhancement of synaptic strength that can be produced by pairing of presynaptic activity with postsynaptic depolarization.

- Long-lasting forms of learning-related synaptic plasticity require transcription and yet occur in a synapse-specific manner, indicating that there are mechanisms to target the products of gene expression to some but not other synapses of a given cell.

- Formation of glutamatergic synapses entails development of "silent" immature contacts into mature functional synapses. To determine how this transformation occurs, we investigated the development of neurotransmission at single synapses in vitro.